record state
frontier-ownedReview status
This finding is part of accepted frontier state. Review events, reviewable changes, and proof state explain how it can change.
unreviewed
frontiers / frontier
Finding bundle
back to stateno incoming links yet
record state
frontier-ownedThis finding is part of accepted frontier state. Review events, reviewable changes, and proof state explain how it can change.
finding statement
finding typeNo entity list is declared.
evidence
source-boundtheoretical · manual state transition
proof impact
packet context1 reviewable changes and 0 evaluation records are attached to this finding id.
Evidence and conditions
method
manual state transition
evidence type
theoretical
conditions
Provenance
source title
Khairkhah et al. 2026 (Neoplasia immunopeptidomics study)
authors
reviewer:will-blair
DMG with H3K27M mutations show enriched H2B1K-derived immunopeptides, revealing histone turnover as a disease mechanism; targeted therapy increases immunopeptide load in DMG but decreases it in GBM.
vs_d7792fa27c780bd9 · manual_curation
outgoing
vf_6d815c2d1decc76dH2B1K immunopeptide enrichment in DMG reflects H3K27M-driven histone turnover; MMR deficiency may further dysregulate this proteostatic balance
incoming
No incoming links.
events
vev_8c2665cb67a2ffd1finding.assertedManual finding added to frontier state
reviewer:will-blair · 2026-05-29
reviewable changes
vpr_1bf838329353d279finding.addManual finding added to frontier state
applied · reviewer:will-blair · 2026-05-29
evaluations
No evaluation record targets this finding id.